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Post by n00b on Sept 3, 2020 12:40:18 GMT -5
This paper reports that PSU's director of Athletic Medicine claimed at a meeting on Monday evening that 30-40% of student athletes infected with COVID (whether symptomatic or not) displayed evidence of myocarditis on MRI scans. He's uncertain how this will play out over the long term, but it's worrying and is likely to influence athlete performance at a minimum. www.centredaily.com/sports/college/penn-state-university/psu-football/article245448050.htmlThat seems like a really high number, but I'm not up on this issue. It may be that no one thinks to do MRIs on anyone other than athletes. It is known that the gene for the external cellular receptor (ACE2) that Coronavirus uses to get into cells is highly expressed in cardiac tissue - more so than in lung tissue. Also, nobody is saying athletes with active COVID should be playing.
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Post by n00b on Sept 3, 2020 12:46:51 GMT -5
Ok, so how to you say the above but allow students on campus and allow teams to practice. Setting the line at intercollegiate competition seems like a very arbitrary line. Athletes would've had multiple COVID tests in the week leading up to the games. What is the evidence that there is any sort of significant risk in competitive athletics? Also, only the NBA and NHL are in bubbles. MLB, MLS, all European soccer, and soon the NFL are not in bubbles. That line doesn’t seem arbitrary to me, if you consider that disallowing competitions prevents cross-contamination between teams. The thing to always keep in mind here is: the world isn’t binary. Taking some measure, which doesn’t reduce the problems by 100%, but does reduce them by even a small amount, is still a valid measure. We can’t have all or nothing logic, on this. That isn’t valid. Totally agree that it isn't binary. Generally having students on campus seems to be 100x more risky than intercollegiate competition. And daily practices also seem a LOT riskier than football games, which are played outdoors and would create essentially zero CDC-defined contacts between the two teams. (The CDC defines a contact as being within 6 feet of a person for 15 minutes or more)
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Post by Deleted on Sept 3, 2020 12:48:09 GMT -5
The decision to play isn’t Warrens, it’s the University Presidents. I’m sure the Big Ten wants to play. This. It amazes me that everyone doesn't yet understand the role of a commissioner. He works for the schools. The presidents are his superiors, not the other way around. His job, like other sports commissioners (Roger Goodell et al) is to act as a mouthpiece and firewall for the presidents. If, as a fan, you're mad at a commissioner and not the owners/presidents, then he's doing his job well and you're falling for the misdirection.
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Post by badgerbreath on Sept 3, 2020 12:49:01 GMT -5
This paper reports that PSU's director of Athletic Medicine claimed at a meeting on Monday evening that 30-40% of student athletes infected with COVID (whether symptomatic or not) displayed evidence of myocarditis on MRI scans. He's uncertain how this will play out over the long term, but it's worrying and is likely to influence athlete performance at a minimum. www.centredaily.com/sports/college/penn-state-university/psu-football/article245448050.htmlThat seems like a really high number, but I'm not up on this issue. It may be that no one thinks to do MRIs on anyone other than athletes. It is known that the gene for the external cellular receptor (ACE2) that Coronavirus uses to get into cells is highly expressed in cardiac tissue - more so than in lung tissue. Also, nobody is saying athletes with active COVID should be playing. I think the worry is that the inflammation may extend beyond the infection period if it is damage caused by the immune response, or an autoimmune response. No one really knows yet.
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Post by JT on Sept 3, 2020 12:50:10 GMT -5
This paper reports that PSU's director of Athletic Medicine claimed at a meeting on Monday evening that 30-40% of student athletes infected with COVID (whether symptomatic or not) displayed evidence of myocarditis on MRI scans. He's uncertain how this will play out over the long term, but it's worrying and is likely to influence athlete performance at a minimum. www.centredaily.com/sports/college/penn-state-university/psu-football/article245448050.htmlThat seems like a really high number, but I'm not up on this issue. It may be that no one thinks to do MRIs on anyone other than athletes. It is known that the gene for the external cellular receptor (ACE2) that Coronavirus uses to get into cells is highly expressed in cardiac tissue - more so than in lung tissue. Also, nobody is saying athletes with active COVID should be playing. True, but irrelevant. Myocarditis Effects lasts beyond Covid-19 being active/infectious. www.si.com/college/2020/08/09/ncaa-cardiac-inflamation-coronavirus-myocarditis-concernsAt least one college football player has developed an enlarged heart after contracting COVID-19, a team trainer told SI under the condition of anonymity. The recovery time for such heart damage is a minimum of three months of no activity, says Martinez.
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Post by volleav on Sept 3, 2020 12:50:21 GMT -5
This paper reports that PSU's director of Athletic Medicine claimed at a meeting on Monday evening that 30-40% of student athletes infected with COVID (whether symptomatic or not) displayed evidence of myocarditis on MRI scans. He's uncertain how this will play out over the long term, but it's worrying and is likely to influence athlete performance at a minimum. www.centredaily.com/sports/college/penn-state-university/psu-football/article245448050.htmlThat seems like a really high number, but I'm not up on this issue. It may be that no one thinks to do MRIs on anyone other than athletes. It is known that the gene for the external cellular receptor (ACE2) that Coronavirus uses to get into cells is highly expressed in cardiac tissue - more so than in lung tissue. Why are athletes the only ones who seem to develop this or are all the media seems concerned with??? I mean not a single person is concerned about anyone but those trying to play college sports, especially football. I'm pretty sure they should be concerned with the Rock. He looks kinda athletic. Or maybe the runners who just do that for fun. Or just anyone who works out.
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Post by oldman on Sept 3, 2020 12:50:38 GMT -5
This paper reports that PSU's director of Athletic Medicine claimed at a meeting on Monday evening that 30-40% of student athletes infected with COVID (whether symptomatic or not) displayed evidence of myocarditis on MRI scans. He's uncertain how this will play out over the long term, but it's worrying and is likely to influence athlete performance at a minimum. www.centredaily.com/sports/college/penn-state-university/psu-football/article245448050.htmlThat seems like a really high number, but I'm not up on this issue. It may be that no one thinks to do MRIs on anyone other than athletes. It is known that the gene for the external cellular receptor (ACE2) that Coronavirus uses to get into cells is highly expressed in cardiac tissue - more so than in lung tissue. Also, nobody is saying athletes with active COVID should be playing. Pre Covid article about Flu and myocarditis. So if you have the flu you can get myocarditis. Do you know that? Its even at the same percentage as Covid. healthblog.uofmhealth.org/heart-health/when-a-virus-turns-deadly-what-you-should-know-about-myocarditis
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Post by n00b on Sept 3, 2020 12:51:05 GMT -5
The decision to play isn’t Warrens, it’s the University Presidents. I’m sure the Big Ten wants to play. This. It amazes me that everyone doesn't yet understand the role of a commissioner. He works for the schools. The presidents are his superiors, not the other way around. His job, like other sports commissioners (Roger Goodell et al) is there to act as a mouthpiece and firewall for the presidents. If, as a fan, you're mad at a commissioner and not the owners/presidents, then he's doing his job well and you're falling for the misdirection. I agree. But there's a reason Warren is getting slammed but not Larry Scott (at least about this). It IS Warren's job to mediate the conversation, whether it's an issue at the president level or the AD level. If that meeting was adjourned without his assurance that all 14 presidents were going to buy in (even if they voted against), AND they had to get their ADs and football coaches to buy in, THAT is Warren's failure.
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Post by Deleted on Sept 3, 2020 12:55:34 GMT -5
This. It amazes me that everyone doesn't yet understand the role of a commissioner. He works for the schools. The presidents are his superiors, not the other way around. His job, like other sports commissioners (Roger Goodell et al) is there to act as a mouthpiece and firewall for the presidents. If, as a fan, you're mad at a commissioner and not the owners/presidents, then he's doing his job well and you're falling for the misdirection. I agree. But there's a reason Warren is getting slammed but not Larry Scott (at least about this). It IS Warren's job to mediate the conversation, whether it's an issue at the president level or the AD level. If that meeting was adjourned without his assurance that all 14 presidents were going to buy in (even if they voted against), AND they had to get their ADs and football coaches to buy in, THAT is Warren's failure. No. It's his job to tell everyone the decision and be the piñata for folks like you. The presidents f'ed this up because they couldn't find a consensus OR keep their people in line. Neither of those are Warren's responsibilities. He's doing a first class job of keeping the masses distracted and shielding the presidents from your ire. He deserves a raise tbh!
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Post by JT on Sept 3, 2020 13:00:06 GMT -5
This paper reports that PSU's director of Athletic Medicine claimed at a meeting on Monday evening that 30-40% of student athletes infected with COVID (whether symptomatic or not) displayed evidence of myocarditis on MRI scans. He's uncertain how this will play out over the long term, but it's worrying and is likely to influence athlete performance at a minimum. www.centredaily.com/sports/college/penn-state-university/psu-football/article245448050.htmlThat seems like a really high number, but I'm not up on this issue. It may be that no one thinks to do MRIs on anyone other than athletes. It is known that the gene for the external cellular receptor (ACE2) that Coronavirus uses to get into cells is highly expressed in cardiac tissue - more so than in lung tissue. Why are athletes the only ones who seem to develop this or are all the media seems concerned with??? I mean not a single person is concerned about anyone but those trying to play college sports, especially football. I'm pretty sure they should be concerned with the Rock. He looks kinda athletic. Or maybe the runners who just do that for fun. Or just anyone who works out. There was an earlier study that showed cardiac damage/stress in (iirc) more than 70% of people, including mild and asymptomatic cases. I posted it in one of the threads, and among other complaints, people said that 100 was too small a group to be significant.
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Post by JT on Sept 3, 2020 13:04:41 GMT -5
Didn’t see anything in the article regarding frequency of influenza-caused myocarditis. Also, we have a vaccine (not 100% effective) for influenza. We have some level of herd immunity even without a vaccine, because we’ve had influenza strains floating around the human population for longer than anyone’s been alive. We have known, understood, and generally reliable treatments for influenza.
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Post by n00b on Sept 3, 2020 13:05:45 GMT -5
Why are athletes the only ones who seem to develop this or are all the media seems concerned with??? I mean not a single person is concerned about anyone but those trying to play college sports, especially football. I'm pretty sure they should be concerned with the Rock. He looks kinda athletic. Or maybe the runners who just do that for fun. Or just anyone who works out. There was an earlier study that showed cardiac damage/stress in (iirc) more than 70% of people, including mild and asymptomatic cases. I posted it in one of the threads, and among other complaints, people said that 100 was too small a group to be significant. This is the study that the Michigan cardiologist said was totally bogus.
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Post by Deleted on Sept 3, 2020 13:07:24 GMT -5
This paper reports that PSU's director of Athletic Medicine claimed at a meeting on Monday evening that 30-40% of student athletes infected with COVID (whether symptomatic or not) displayed evidence of myocarditis on MRI scans. He's uncertain how this will play out over the long term, but it's worrying and is likely to influence athlete performance at a minimum. www.centredaily.com/sports/college/penn-state-university/psu-football/article245448050.htmlThat seems like a really high number, but I'm not up on this issue. It may be that no one thinks to do MRIs on anyone other than athletes. It is known that the gene for the external cellular receptor (ACE2) that Coronavirus uses to get into cells is highly expressed in cardiac tissue - more so than in lung tissue. Why are athletes the only ones who seem to develop this or are all the media seems concerned with??? I mean not a single person is concerned about anyone but those trying to play college sports, especially football. I'm pretty sure they should be concerned with the Rock. He looks kinda athletic. Or maybe the runners who just do that for fun. Or just anyone who works out. They aren't the only ones who develop this. But, most everything posted in here is from a sports perspective so focusing on athletes is a pretty obvious angle to take. Plus, if I develop myocarditis if I were to get COVID I am not going to spend several hours a day with massive physical exertion like an athlete would. I may go for a walk or even a run, but my job is sitting at a desk. Not running as fast as I can and colliding with other people. People would have been concerned with The Rock back when he was a football player at The U. He is a grown ass man and makes decisions for himself and only impact himself. Presidents have to make decisions that impact many.
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Post by badgerbreath on Sept 3, 2020 13:09:32 GMT -5
This paper reports that PSU's director of Athletic Medicine claimed at a meeting on Monday evening that 30-40% of student athletes infected with COVID (whether symptomatic or not) displayed evidence of myocarditis on MRI scans. He's uncertain how this will play out over the long term, but it's worrying and is likely to influence athlete performance at a minimum. www.centredaily.com/sports/college/penn-state-university/psu-football/article245448050.htmlThat seems like a really high number, but I'm not up on this issue. It may be that no one thinks to do MRIs on anyone other than athletes. It is known that the gene for the external cellular receptor (ACE2) that Coronavirus uses to get into cells is highly expressed in cardiac tissue - more so than in lung tissue. Why are athletes the only ones who seem to develop this or are all the media seems concerned with??? I mean not a single person is concerned about anyone but those trying to play college sports, especially football. I'm pretty sure they should be concerned with the Rock. He looks kinda athletic. Or maybe the runners who just do that for fun. Or just anyone who works out. It's widely recognized as a problem in COVID and other coronavirus infections, but is mostly looked at in clinical settings with active disease and pathology. Athletes just get more preliminary care because their performance depends on peak VO2, and they may be at a higher risk of developing arrhythmias when heart rate is elevated during exercise. I've read perspectives from a few runners complaining of longer term shortness of breath and elevated heart rates after COVID.
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Post by badgerbreath on Sept 3, 2020 13:21:18 GMT -5
Yes. I don't know if the number quoted is statistically dependable, but 30-35 % is quite a bit more than typically reported for influenza, which is 10%. Plus, influenza is not as rampant because vaccine, history of exposure, less efficient spread, etc, so that reduces the population incidence of influenza caused myocarditis even more. Finally, we just don't know much about COVID and the kind of damage it can produce. It's only been around for 8-10 months.
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